F&N 4-0

12/02

 

Abstract

Acrylamide

 

 

Prepared by:   Barbara Brown, Ph.D., R.D./L.D.

                        Food Specialist

                        104 HES, Room 321

                        OCES, Oklahoma State University

                        Stillwater, OK 74078-6141

                        405-744-6824

                        bbrown@okstate.edu

 


IMPLICATIONS FOR COOPERATIVE EXTENSION.  The potential carcinogen acrylamide has been found in a wide variety of food favorites of many Oklahomans.  However, until more is known, including whether or not acrylamide is a human carcinogen, it is important to continue to eat a balanced diet high in fruits, vegetables, and whole grains.  People should focus on diets lower in foods high in acrylamide.  That would mean limiting the amount of fried potato and snack foods eaten.

 

 

In April, 2002, researchers at the Swedish National Food Administration and Stockholm University reported finding the chemical acrylamide in a variety of fried and oven-baked foods. The initial Swedish research indicates that acrylamide formation is particularly associated with traditional high temperature cooking processes for certain carbohydrate-rich foods. Since the Swedish report, similar findings have been reported by Norway, the United Kingdom, and Switzerland. Recently, FDA developed a test protocol for reliably quantifying acrylamide concentrations in food. It has used it to preliminarily confirm what government agencies in Sweden and several other European countries reported earlier this year—that it is in a broad range of cooked foods, especially breads and potato products. The discovery is a concern because acrylamide is a potential human carcinogen.

 

Acrylamide appears to form as a byproduct of high-temperature cooking (greater than 248°F). It does not appear to be present in food before cooking. At this time, not enough is known about its formation to identify safe changes to food processing that will clearly prevent or reduce formation. Understanding how it is formed will be an important step in identifying ways to reduce or prevent formation during cooking.

 

People have been eating some of the foods now reported to contain acrylamide for many years. To better assess the risk more information is needed about which foods acrylamide is formed in, levels of acrylamide in foods, dietary exposure, the bioavailability of acrylamide in food, the potential of acrylamide to cause cancer when eaten in food, and biomarkers of acrylamide exposure.

 

Acrylamide causes cancer in laboratory animals. However, it is not clear whether it causes cancer in humans. Scientists have conducted epidemiological studies of people exposed to acrylamide in the workplace. The studies did not show increased cancer risk with acrylamide exposure. However, these studies do not rule out the possibility that acrylamide in food can cause cancer, both because of the limited number of people in the studies and because the route of exposure for the workers was not through food.

 

Addressing the issue of acrylamide in foods will require research and activity on multiple fronts. Planned and current actions by the FDA include assessing the dietary exposure of U.S. consumers to acrylamide, gathering new information about the toxicology of acrylamide, and participating in the development of techniques for reducing acrylamide


Acrylamide (continued)

 

 


formation. As more data become available on acrylamide, FDA will continue to develop public health messages and regulatory options.

 

The agency plans a “total diet study” using store-bought samples of foods. The findings, weighted to reflect the quantities of each food consumed, will be summed to estimate an individual’s typical daily intake of acrylamide.

 

A preliminary assessment of human exposures was presented in September by the Center for Food Nutrition Policy at the Virginia Polytechnic Institute and State University in Alexandria. The center’s indicates for adults, likely daily consumption of acrylamide from food runs from 0.5 to 0.8 micrograms per kilogram of body weight. For children, average exposure is probably double that. However, people whose diets favor the most heavily tainted foods could eat as much as 6.2 µg/kg daily, and children could consume up to 18 µg/kg each day.

 

These estimates all exceed the EPA’s 0.2 µg/kg per day recommended limit for consumption in drinking water. However EPA has said that the impact of consumption at even their low limit remains unknown.

In an attempt to resolve acrylamide’s risk, FDA will begin short-term animal tests to compare how much acrylamide the body takes up from drinking water compared to food. If the availability of the compound from these two sources proves similar, then existing toxicological data—performed to evaluate acrylamide’s use in water-treatment plants—can be used to gauge impacts from low-dose exposures in food. If the body takes up acrylamide more readily from food than from water, then new studies may be needed.

 

References:

Raloff, J. (2002) FDA launches acrylamide investigations. Science News Online. Volume 162, Number 15 (October 12). http://www.sciencenews.org/20021012/food.asp

CSPI. (2002) New tests confirm acrylamide in American foods. Center for Science in the Public Interest press release. June 25.  http://www.cspinet.org/new/200206251.html.

Food and Drug Administration. (2002) Assessing acrylamide in the U.S. food supply; public meeting; draft action plan on acrylamide; availability. Federal Register 67 (Sept. 12): 57827-57828. http://www.cfsan.fda.gov/~lrd/fr020912.html